RORα is critical for mTORC1 activity in T cell-mediated colitis

Targeting pro-inflammatory T cells as a novel therapeutic approach to potentially resolve atherosclerosis in humans

RORα is critical for mTORC1 activity in T cell-mediated colitis - ScienceDirect

IJMS, Free Full-Text

清华大学免疫学研究所董晨课题组揭示T细胞肠炎致病性新机制- 清华大学免疫学研究所

Wei JIN, Co-PI/Research Track Associate Professor, Doctor of Philosophy, Tsinghua University, Beijing, TH, Institute for Immunology and School of Medicine

RORα overexpression reduced interleukin‐33 expression and prevented mast cell degranulation and inflammation by inducing autophagy in allergic rhinitis - Yu - 2023 - Immunity, Inflammation and Disease - Wiley Online Library

The neuroimmune axis of Alzheimer's disease, Genome Medicine

WO2018053109A1 - Gene expression characterization of a glatiramer acetate related drug product in mammalian and human cells - Google Patents

IL-7Rα blockade efficiency in vivo in humanized acute TNBS colitis. (A

RORα suppresses Th17-related inflammation

The neuroimmune axis of Alzheimer's disease, Genome Medicine

RORA regulates neutrophil migration and activation in zebrafish

RORα is critical for mTORC1 activity in T cell-mediated colitis - ScienceDirect

Advantageous tactics with certain probiotics for the treatment of graft-versus-host-disease after hematopoietic stem cell transplantation

Colonic epithelial mTORC1 promotes ulcerative colitis through COX-2-mediated Th17 responses - ScienceDirect